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anti sara  (Proteintech)


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    Structured Review

    Proteintech anti sara
    Anti Sara, supplied by Proteintech, used in various techniques. Bioz Stars score: 88/100, based on 6 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/anti+sara/SARA+Antibody/10__1096_slash_fj__202503305r-94-7-13
    Average 88 stars, based on 6 article reviews
    anti sara - by Bioz Stars, 2026-11
    88/100 stars

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    Related Articles

    other:

    Article Title: Structural Basis for Specific Interaction of TGFβ Signaling Regulators SARA/Endofin with HD-PTP
    Article Snippet: Rabbit: anti-HD-PTP (Proteintech Cat. No.102472-1-AP), anti-endofin (Proteintech Cat. No. 13118-2-AP), anti-SARA (Proteintech Cat. No. 22033-1-AP).

    Article Title: SARA suppresses myofibroblast precursor transdifferentiation in fibrogenesis in a mouse model of scleroderma
    Article Snippet: Antibodies used were; anti-NG2 (MA5- 24247, ThermoFisher, Waltham, MA) (1:75), anti-aSMA (ab5694, abcam, Cambridge, UK) (1:150), anti-CD31-Alexa647 (102416, BioLegend, San Diego, CA) (1:75), anti-CD45 (ab23910, abcam, Cambridge, UK) (1:100), anti-CD146-APC (50-1469-42, ThermoFisher, Waltham, MA) (1:40), antiIL-31 (MA5-34912, ThermoFisher, Waltham, MA), anti-pSTAT3 (ab267373, abcam, Cambridge, UK), anti-SARA (14821-1- AP, Proteintech, Rosemont, IL), anti-MFAP5 (15727-1-AP, Proteintech, Rosemont, IL) and anti-CTHRC1 (16534-1-AP, Proteintech, Rosemont, IL) .

    Article Title: <scp>Parthenolide</scp> Alleviates Peritoneal Fibrosis by Blocking Smad2/3 Phosphorylation via Smad Anchor for Receptor Activation
    Article Snippet: Progressive peritoneal fibrosis is a severe complication of peritoneal dialysis (PD) with a complex pathogenesis.. Our previous studies demonstrated that parthenolide (PTL) alleviates peritoneal fibrosis by suppressing the transforming growth factor (TGF)β/ Smad pathway.. Smad anchor for receptor activation (SARA) acts as an adaptor protein for Smad2 and Smad3; however, its role in PDassociated peritoneal fibrosis and the relationship between PTL and SARA remain to be elucidated.

    Article Title: Structural Basis for Specific Interaction of TGFβ Signaling Regulators SARA/Endofin with HD-PTP
    Article Snippet: anti-SARA , Proteintech , RRID: AB_11182938.

    Incubation:

    Article Title: Dachshund Depletion Disrupts Mammary Gland Development and Diverts the Composition of the Mammary Gland Progenitor Pool
    Article Snippet: .. For each IP, 1 mL lysate (1 mg protein) and 2 μg anti-SMAD2/3 (BD, cat. no. 610843) or anti-SARA (Proteintech, cat. no. 14821-1-AP) were incubated overnight at 4°C. ..



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    anti sara  (Proteintech)
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    Figure 3. Effects of E7050 on VEGFR2-mediated signaling pathways in VEGF-stimulated HUVECs. Cells were serum-starved for 6 h and then pretreated with E7050 (5 and 10 µM) or vehicle for 1 h, followed by stimulation with VEGF (100 ng/mL) for another 10 min <t>(VEGFR2,</t> <t>PLCγ1,</t> <t>FAK,</t> and Src)
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    Figure 3. Effects of E7050 on VEGFR2-mediated signaling pathways in VEGF-stimulated HUVECs. Cells were serum-starved for 6 h and then pretreated with E7050 (5 and 10 µM) or vehicle for 1 h, followed by stimulation with VEGF (100 ng/mL) for another 10 min (VEGFR2, PLCγ1, FAK, and Src)

    Journal: International journal of molecular sciences

    Article Title: E7050 Suppresses the Growth of Multidrug-Resistant Human Uterine Sarcoma by Inhibiting Angiogenesis via Targeting of VEGFR2-Mediated Signaling Pathways.

    doi: 10.3390/ijms24119606

    Figure Lengend Snippet: Figure 3. Effects of E7050 on VEGFR2-mediated signaling pathways in VEGF-stimulated HUVECs. Cells were serum-starved for 6 h and then pretreated with E7050 (5 and 10 µM) or vehicle for 1 h, followed by stimulation with VEGF (100 ng/mL) for another 10 min (VEGFR2, PLCγ1, FAK, and Src)

    Article Snippet: Primary antibodies against VEGFR2 (#2479; 1:20,000 dilution), phospho (p)-VEGFR2 (Tyr1175) (#2478; 1:1500 dilution), Akt (#4691; 1:20,000 dilution), p-Akt (Ser473) (#4060; 1:5000 dilution), FAK (#3285; 1:7000 dilution), p-FAK (Tyr397) (#8586; 1:1500 dilution), PLCγ1 (#5690; 1:8000 dilution), p-PLCγ1 (Ser1248) (#8713; 1:2500 dilution), Src (#2109; 1:30,000 dilution), p-Src (Tyr416) (#6943; 1:6000 dilution), JNK (#9258; 1:1000 dilution), pJNK (Thr183/Tyr185) (#4668; 1:1000 dilution), ERK1/2 (#4695; 1:15,000 dilution), p-ERK1/2 (Thr202/Tyr204) (#4370; 1:15,000 dilution), p38 MAPK (#9212; 1:12,000 dilution), and pp38 MAPK (Thr180/Tyr182) (#4511; 1:2000 dilution) were purchased from Cell Signaling Technology (Beverly, MA, USA).

    Techniques: Protein-Protein interactions

    Figure 7. Schematic diagram of a proposed mechanism of E7050-induced anti-angiogenic activity. E7050 exerts anti-angiogenic effects in VEGF-stimulated endothelial cells by downregulating the phosphorylation of VEGFR2 and its downstream mediators, including PLCγ1, FAK, Src, Akt, JNK, and p38 MAPK. The blockage of VEGFR2-mediated signaling cascade pathways by E7050 contributes to the inhibition of proliferation, migration, and tube formation in endothelial cells.

    Journal: International journal of molecular sciences

    Article Title: E7050 Suppresses the Growth of Multidrug-Resistant Human Uterine Sarcoma by Inhibiting Angiogenesis via Targeting of VEGFR2-Mediated Signaling Pathways.

    doi: 10.3390/ijms24119606

    Figure Lengend Snippet: Figure 7. Schematic diagram of a proposed mechanism of E7050-induced anti-angiogenic activity. E7050 exerts anti-angiogenic effects in VEGF-stimulated endothelial cells by downregulating the phosphorylation of VEGFR2 and its downstream mediators, including PLCγ1, FAK, Src, Akt, JNK, and p38 MAPK. The blockage of VEGFR2-mediated signaling cascade pathways by E7050 contributes to the inhibition of proliferation, migration, and tube formation in endothelial cells.

    Article Snippet: Primary antibodies against VEGFR2 (#2479; 1:20,000 dilution), phospho (p)-VEGFR2 (Tyr1175) (#2478; 1:1500 dilution), Akt (#4691; 1:20,000 dilution), p-Akt (Ser473) (#4060; 1:5000 dilution), FAK (#3285; 1:7000 dilution), p-FAK (Tyr397) (#8586; 1:1500 dilution), PLCγ1 (#5690; 1:8000 dilution), p-PLCγ1 (Ser1248) (#8713; 1:2500 dilution), Src (#2109; 1:30,000 dilution), p-Src (Tyr416) (#6943; 1:6000 dilution), JNK (#9258; 1:1000 dilution), pJNK (Thr183/Tyr185) (#4668; 1:1000 dilution), ERK1/2 (#4695; 1:15,000 dilution), p-ERK1/2 (Thr202/Tyr204) (#4370; 1:15,000 dilution), p38 MAPK (#9212; 1:12,000 dilution), and pp38 MAPK (Thr180/Tyr182) (#4511; 1:2000 dilution) were purchased from Cell Signaling Technology (Beverly, MA, USA).

    Techniques: Activity Assay, Phospho-proteomics, Inhibition, Migration